Metabolic alterations related to T2D are well characterised by epidemiological and research primarily based experiments. The pathogenesis and progression of T2D is ascribed to four mechanisms; improved Sophisticated glycation conclusion products (AGE) development, elevated polyol pathway flux, activation of protein kinase C (PKC) isoforms, and elevated hexosamine pathway flux[7]. Until https://www.directivepublications.org/journal-of-diabetology-research/